Stanozolol abuse can also cause increased bromsulphalein (BSP) retention, and increases in serum bilirubin, glutamic oxaloacetic transaminase (SGOT), and alkaline phosphatase or inhibition of testicular function, testicular atrophy and oligospermia, impotence, chronic priapism, epididymitis and bladder irritability, edema, retention of serum electrolytes (sodium, chloride, potassium, phosphate, and calcium). Abuse of this drug can even cause increased or decreased libido, habituation, excitation, insomnia, depression, bleeding in patients on concomitant anticoagulant therapy, premature closure of epiphyses in children, nausea, vomiting, and diarrhea.
Because the ultimate goal of a steroid cycle is to increase strength and muscle size, the associated spike in estrogen which accompanies steroids such as Testosterone is considered undesirable. In order to disassociate the two effects, two classes of drug are used. Medications such as Nolvadex or Clomid target the estrogen receptors. They make it more difficult for the estrogen to exert it’s influence within the body thus allowing the testosterone to act more freely. The second class is aromatase inhibitors such as Femara. They target the aromatase enzyme itself in order to prevent the production of estrogen in the first place. Sometimes, it’s not always clear which option you should go with or even what the differences are between the two. Lets clear that up a little.